RETINAL ATROPHIC HOLE FULL
RETINAL ATROPHIC HOLE FREE

6 - 10 In such cases, the surgical success rate was suboptimal because the detection of retinal breaks is critical for the successful treatment of retinal detachments. However, retinal detachments with undetected breaks have been frequently reported in the literature. Retinal detachment represents a major vision-threatening complication of pathologic myopia and is associated with peripheral retinal tears or macular holes. 2 - 5 Detection of these retinal pathologic conditions helps to understand or explain the progressive degenerative changes of pathologically myopic eyes. Recently, other abnormalities affecting the posterior pole have been described, such as paravascular retinal microfolds, intraretinal cysts, lamellar holes, inner retinal cleavage, and foveoschisis. 1 Various sequela, such as staphyloma, macular hole, and chorioretinal atrophy, have been widely recognized. It is characterized by excessive axial elongation with progressive degeneration of the posterior pole. Pathologic myopia is one of the leading causes of blindness in the world. Pars plana vitrectomy with photocoagulation and intraocular tamponade may lead to a resolution of such rhegmatogenous retinal detachments. These breaks are apt to elude detection before surgery, and a careful search along the posterior vascular arcades during vitrectomy may help to detect these abnormalities. Vitrectomy with gas tamponade was performed in all cases, and retinal reattachment was achieved in 9 cases by a single operation.Ĭonclusions Paravascular linear retinal breaks over areas of patchy chorioretinal atrophy represent a distinct clinical entity that can result in a special category of retinal detachment among patients with pathologic myopia. These paravascular linear retinal breaks have distinct clinical characteristics, including a strong propensity to occur over areas of patchy chorioretinal atrophy, a linear shape that is oriented parallel to the adjacent retinal vessels, and a tendency to result in progressive retinal detachment. The length of the breaks ranged from 0.25 to 1 disc diameter, and their distance from the optic disc ranged from 1 to 5 disc diameters. The retinal breaks were most frequently found along the inferotemporal vascular arcade, followed by the superotemporal arcade. Results Ten eyes with posterior paravascular linear retinal breaks and retinal detachment were identified from January 1, 2008, to July 31, 2009. Methods In this retrospective case series, we evaluated 10 pathologically myopic eyes having rhegmatogenous retinal detachment associated with posterior paravascular linear retinal breaks. Objective To characterize posterior paravascular linear retinal breaks over areas of patchy chorioretinal atrophy as a cause of retinal detachment among patients with pathologic myopia. Shared Decision Making and Communication.Scientific Discovery and the Future of Medicine.Health Care Economics, Insurance, Payment.

Clinical Implications of Basic Neuroscience.

Challenges in Clinical Electrocardiography.

Therefore treatment must be tailored to specific situations, bearing in mind that the benefits of treatment must be weighted against potential risks. The vast majority of retinal breaks do not cause retinal detachment. They are usually found in the infero-temporal retina.Ītrophic holes are caused by atrophy of the inner retinal layers and are not associated with vitreoretinal traction.

RETINAL ATROPHIC HOLE FREE

Operculated holes are also caused by vitreoretinal traction, that is usually stronger and released during the hole formation (vitreous remains attached to the operculum and free from attached retina).ĭialyses are circumferential linear breaks occurring either at the anterior or posterior margin of the vitreous base. Tears are most commonly found in the supero-temporal quadrant. When tears exceed 90 degrees circumferentially they are called giant tears. They are caused by vitreous traction, exerted on the flap (frequently the vitreous remains attached to the flap and traction persists). Horseshoe tears, have a U configuration, with the convexity usually pointing to the optic disc. According to their configuration breaks can be divided in: horseshoe tears, giant tears, operculated holes, dialyses, and atrophic retinal holes. When significant vitreoretinal traction is present at the break site and a liquefied vitreous is present, a retinal detachment can occur.īreaks can be caused by atrophy of the inner retinal layers, vitreoretinal traction or trauma.

RETINAL ATROPHIC HOLE FULL

Retinal breaks are full thickness defects of the neurosensorial retina.

Retinal atrophic hole